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Dissertationen der Medizinischen Fakultät

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Browse by Advisor: Kuner, Prof. Dr. Rohini

Jump to: 2024 | 2022 | 2021 | 2019 | 2017 | 2014 | 2013
Number of items: 14.

2024

Christiane, Höper (2024) Molekulare Mechanismen zwischen Tumorzellen und Neuronen beim duktalen Adenokarzinom des Pankreas sowie ihr Einfluss auf neurale Invasion im Hinblick auf Tumor-induzierten Schmerz.

Gan, Zheng (2024) Understanding the nature and function of pathways to improve brain stimulation as a therapeutic strategy in chronic pain.

2022

Li, Han (2022) Analysis of transcranial cortical stimulation in mice to alleviate neuropathic pain.

Liu, Sheng (2022) Spinal cord circuits underlying dynamic and punctate allodynia in inflammatory pain & Cellular basis for prefrontal modulation of inflammatory pain by fear.

2021

Ding, Ting (2021) Study on the role of BDNF-TrkB signaling in chronic neuropathic pain.

Wang, Na (2021) Functional changes in primary afferent fibers in diabetic peripheral neuropathy in mice.

2019

Arcourt, Alice (2019) Touch receptor inputs alleviate acute pain signalling and are essential for nociceptive reflex coordinationt.

Moll, Martin (2019) Nerve Injury-Induced Neuronal Cell Death In The Dorsal Horn Of The Spinal Cord Contributes To Chronic Neuropathic Pain.

Prato, Vincenzo (2019) Functional properties and molecular markers of mechanoinsensitive “silent” nociceptors.

2017

Gritsch, Simon (2017) Mechanisms underlying central pain in animal models of multiple sclerosis and stroke.

2014

Lu, Jianning (2014) Mechanisms underlying structural and functional plasticity in mouse spinal cord in chronic pain states.

Njoo, Christian (2014) Proteomics-based identification of interaction partners of cannabinoid receptor 1 and analyses of their roles in structural and functional plasticity of neurons.

2013

Bali, Kiran Kumar (2013) Genome-wide Identification and functional validation of microRNAs and mRNAs in chronic pain.

Gangadharan, Vijayan (2012) Glutamatergic and GABAergic signaling in peripheral nociceptive neurons: contributions to chronic pain.