title: RAGE does not contribute to renal injury and damage upon ischemia/reperfusion-induced injury
creator: Dessing, Mark C.
creator: Pulskens, Wilco P.
creator: Teske, Gwendoline J.
creator: Butter, Loes M.
creator: Poll, Tom van der
creator: Yang, Huan
creator: Tracey, Kevin J.
creator: Nawroth, Peter P.
creator: Bierhaus, Angelika
creator: Florquin, Sandrine
creator: Leemans, Jaklien C.
subject: 610
subject: 610 Medical sciences Medicine
description: The receptor for advanced glycation end products (RAGE) mediates a variety of inflammatory responses in renal diseases, but its role in renal ischemia/reperfusion (I/R) injury is unknown. We showed that during renal I/R, RAGE ligands HMGB1 and S100B are expressed. However, RAGE deficiency does not affect renal injury and function upon I/R-induced injury. Copyright (C) 2011 S. Karger AG, Basel
publisher: S. Karger AG
date: 2012
type: Article
type: info:eu-repo/semantics/article
type: NonPeerReviewed
format: application/pdf
identifier: https://archiv.ub.uni-heidelberg.de/volltextserverhttps://archiv.ub.uni-heidelberg.de/volltextserver/17824/1/JIN2012004001080.pdf
identifier: DOI:10.11588/heidok.00017824
identifier: urn:nbn:de:bsz:16-heidok-178245
identifier:   Dessing, Mark C. ; Pulskens, Wilco P. ; Teske, Gwendoline J. ; Butter, Loes M. ; Poll, Tom van der ; Yang, Huan ; Tracey, Kevin J. ; Nawroth, Peter P. ; Bierhaus, Angelika ; Florquin, Sandrine ; Leemans, Jaklien C.  (2012) RAGE does not contribute to renal injury and damage upon ischemia/reperfusion-induced injury.  Journal of innate immunity, 4 (1).  pp. 80-85.  ISSN 1662-811X     
relation: https://archiv.ub.uni-heidelberg.de/volltextserver/17824/
rights: info:eu-repo/semantics/openAccess
rights: http://archiv.ub.uni-heidelberg.de/volltextserver/help/license_urhg.html
language: eng